Kenneth Marcus is INSANE.....

2003-12-21 01:04:18 AM
Subject:
[scilyme] Kenneth Marcus is INSANE....
Date:
Sat, 20 Dec 2003 09:01:57 -0800 (PST)
From:
jan <janmusinski@earthlink.net>
Reply-To:
scilyme@yahoogroups.com
To:
scilyme@yahoogroups.com, jan <janmusinski@earthlink.net>,
scilyme@yahoogroups.com, scilyme@yahoogroups.com,
scilyme@yahoogroups.com,
scilyme@yahoogroups.com, jan <janmusinski@earthlink.net>,
SpinLyme@yahoogroups.com, scilyme@yahoogroups.com,
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jhvanna@optonline.net, mstone@uconn.law.edu, pjb@grotonlaw.com,
pfh1016@aol.com,
jan <janmusinski@earthlink.net>, jan
<janmusinski@earthlink.net>, GregGerber@hotmail.com,
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james.phillips@yale.edu, benjamin.bunney@yale.edu,
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CC:
diana.urban@po.state.ct.us, pamela.mcavay@po.state.ct.us,
johns357@aol.com
www.dmhas.state.ct.us/medicaldir.htm
... to try to tell the court, Kathleen Dickson is dangerously
intelligent.
'Quite flattering, to bring down the Medical Director of the DMHAS, to
declare me insane, and try to have me committed.
Lyme is a brain disease, and Psychotropics are brain damaging.
And this is the SCIENCE, the EVIDENCE, and not the HEARSAY:
www.ncbi.nlm.nih.gov/entrez/query.fcgi
Neurology. 1990 Oct;40(10):1535-40.
Borrelia burgdorferi infection of the brain: characterization of
the organism and response to antibiotics and immune
sera in the mouse model.
Pachner AR, Itano A.
Department of Neurology, Georgetown University Hospital,
Washington, DC 20007.
To learn more about the neurologic involvement in Lyme disease, we
inoculated inbred mice with the causative agent of
Lyme disease, Borrelia burgdorferi. We cultured brains and other
organs, and measured anti-B burgdorferi antibody titers.
We further studied a brain isolate for its plasmid DNA content and its
response in vitro to immune sera and antibiotics.
One strain of B burgdorferi, N40, was consistently infective for mice,
and resulted in chronic infection of the bladder
and spleen. SJL mice developed fewer culture-positive organs and had
lower antibody titers than Balb/c and C57Bl/6 mice.
Organism was cultured from the brain early in the course of infection,
and this isolate, named N40Br, was further studied
in vitro. The plasmid content of N40Br was different from that of the
infecting strain, implying either a highly selective
process during infection or DNA rearrangement in the organism in vivo.
N40Br was very sensitive to antibiotics, but only
after prolonged i!
ncubation. Immune sera from both mice and humans infected with B
burgdorferi were unable to completely kill the organism
by complement-mediated cytotoxicity. These data demonstrate that B
burgdorferi infects the brain of experimental animals,
and is resistant to immune sera in vitro but sensitive to prolonged
treatment with antibiotics.
PMID: 2215944 [PubMed - indexed for MEDLINE]
Genetic animal models: focus on schizophrenia, Gainetdinov, RR., et
al. TRENDS in Neuroscience, Vol 24., No. 9, September,
2001 (They have no clue what the drugs targets are, because the
drugs' mechanisms are all wrong. And that's the state
of the "art".-- KMD)
"Loss of striatal cholinergic neurons as a basis for tardive and
L-dopa-induced dyskinesias, neuroleptic-induced
supersensitivity psychosis and refractory schizophrenia."
Miller R, Chouinard G., Biol Psychiatry. 1993 Nov 15;34(10):713-38.
Psychatric research in the 21st Century; Opportunities and
Limitations, GR, Heninger, Millenium Article, Molecular
Psychiatry (1999) 4, 429-436 (Psychiatry is beginning to consider
thinking like scientists--KMD)
"The Pathophysiology of Agitation", Jearn-Pierre Lindenmayer, J Clin
Psychaitry 2002;61 (suppl 14_
"Akathisia and Exacerbation of Psychopathology; A Preliminary Report",
Dunca, et al. Clinical Neuropharmacology, Vol 23,
No. 3, pp. 169-173
"Subjective Emotional Experioemce and Cognitive Impairment in
Drug-Induced Akathisia", Jong-Hoon Kim, et al, Comprehensive
Psychiatry, Vol.43, No. 6 (November/DEcember), 2002: pp 456-462
"Correlations Between Akathisia and Residual Psychopathology: A
By-product of Neuroleptic-Induced Dysphoria", Newcomer, et
al. Br J Psychiatry. 1994 Jun;164(6):834-8.
"Cardiovascular Effects of Antipsychotics Used in Bipolar Illness",
Piepho, Robert W., J Clin Psychiatry 2002;63 [suppl
4]:20-23
Movement Disorders Associated With Atypical Antipsychotic Drugs,
Caroff, SN, et al, J Clin Psychiatry 2002;63[suppl 4]
12-19
Gamma-aminobutyric acid agonists for neuroleptic-induced dyskensia",
Soares, et al, Cochrane Database Syst Rev 2001;
(2);CD000203
"Traumatic Exposure and PTSD in Borderline, Schizotypal, Avoidant, and
OC Personality Disorders' Findings from the
Collaborative Longitudinal Personality Disorders Study, The Journal of
Nervous and Mental Diseases, Vol 190, No. 8, 2002,
Yale McGlashan et al. (These idiots are beginning to understand that
there are no "personality disorders", there is just
TRAUMA, Organic, and Genetic. No predictive value, only the threat of
misuse of the terminology to the detriment of the
victims of Psychiatry and their pseudopharmaceuticals.-- KMD)
Van Der Kolk Assessment and Treatment of Complex PTSD, Rachel Yehuda,
2001 "Traumatic Stress"
ABSTRACT: www.ncbi.nlm.nih.gov/entrez/query.fcgi
"The relationship of borderline personality disorder to posttraumatic
stress disorder and traumatic events."
Golier JA, Yehuda R, Bierer LM, Mitropoulou V, New AS, Schmeidler J,
Silverman JM, Siever LJ.
Department of Psychiatry, Veterans Affairs Medical Center (116-A), 130
West Kingsbridge Road, Bronx, NY 10468, USA.
julia.golier@med.va.gov CONCLUSIONS: The associations of personality
disorder with early trauma and PTSD were evident,
but modest, in borderline personality disorder and were not unique to
this type of personality disorder. The results do
not appear substantial or distinct enough to support singling out
borderline personality disorder from the other
personality disorders as a trauma-spectrum disorder or variant of
PTSD.
CONCLUSION: There are no axes beyond Genetic, Traumatic, and Organic,
and therefore no DSM diagnosis has any predictive
potential, to the extent that those axes have been studied as such,
framed, and monitored. There is little likelihood
there ever will be, since the datapoints in between appear to be
infinite.
But allow me to quote a Yale Clinical Professor, who knows nothing
about any of those three axes: "...we must recognize
that the practical, individual-directed nature of psychiatric practice
renders psychiatry an inexact science. It is as
inexact as people are different. As practical knowledge, this
inexactness is not a defect that can be overcome with more
progress in the field."--- James Phillips, "Hermeneutics, Key
Concepts, Philosophy, Psychiatry, & Psychology 3.1 (1996)
61-69
It's really much simpler than that off the wall text, and this is the
only statement that has any value, since Psychiatry
is valueless.
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